In AML, the levels of CD123, GM-CSF, interleukin-2 receptor alpha chain (IL-2Rα; CD25), common gamma-chain cytokine (γc), c-kit, and granulocyte colony-stimulating factor receptor (G-CSFR) were widely expressed with a range of more than 10,000 sites/cell, and their expression patterns correlated with morphologic subtypes [36,37]. This evidence concerns the gene CSF3R and acute myeloid leukemia.