Overexpression of FDFT1 as well as its interaction partner, CYP51A1 (cytochrome P-450, family 51, subfamily A), caused an increase in migration/invasion capabilities of cells in vitro and an increase in the number of detectable metastatic lung foci compared with the control animals in vivo, suggesting a positive association between FDFT1, CYP51A1 and invasiveness of lung cancer cells [10]. The gene discussed is CYP51A1; the disease is lung carcinoma.