Moreover, immunohistochemistry assay revealed that downregulation of SEMA3F countervailed the effect of FAM83C-AS1 sponging on the expression of Ki67, PCNA, E-cadherin and N-cadherin, indicating that SEMA3F depletion promoted tumor growth and EMT process (Figure 6F). The gene discussed is MKI67; the disease is neoplasm.