UCA1 and acute myeloid leukemia: Importantly, lncRNA-UCA1 overexpression can reverse the HIF-1α-dependent glycolysis inhibition mediated by miR-125a in HL60andHL60/ADR cells, showing that lncRNA-UCA1 could inhibit glycolysis through miR-125a/HK2 pathway and plays an active role in overcoming chemotherapeutic resistance to AML in children [227].