Notably hepatocyte-specific, pan-splice NCoR knockouts produce a liver steatosis resembling that observed in our NCoRω-/- mice [67]; given this is not observed in our NCoRδ-/- animals it is likely that it is the specific loss of the NCoRω variant that also is responsible for this hepatic phenotype in the pan-splice knockout. Here, NCOR1 is linked to steatosis.