Helminth infections typically induce a T-helper (Th) 2 immune response, with an immunoglobulin E antibody class switch, production and activation of eosinophils, mast cell degranulation [7], and marked elevation of interleukin 4, 5, and 13 (IL-4, IL-5, and IL-13) [6]. Here, IL4 is linked to helminthiasis.