NFE2L2-mediated oxidative stress is a prominent feature of cervical cancer [3], promoting the proliferation, inhibiting the apoptosis, and enhancing the migration and invasion of cervical cancer cells [4, 5], as well as increasing the tumor chemoresistance [6, 7], suggesting that NFE2L2 may be a marker of poor prognosis in cervical cancer patients [8]. This evidence concerns the gene NFE2L2 and neoplasm.