As shown in Figure 6, genes in high-expression groups of CEP55, IFI44, NCF4, and TCIRG1 were all enriched with “Natural killer cell mediated cytotoxicity” and “Primary immunodeficiency” pathways; the “T cell receptor signaling pathway” was enriched in high-expression groups of CEP55, IFI44, and NCF4, whereas the “Intestinal immune network for IgA production” pathway was enriched in CEP55, NCF4, and TCIRG1 high-expression groups, respectively. Here, TCIRG1 is linked to inborn error of immunity.