Although the work of Boettger et al. (2002) and Sarantopoulos et al. (2007) also reported decreased function of KCa3.1 in DRG following nerve injury, the relevance of these findings to pain etiology is questioned by the observation that KCa3.1-/1 mice show normal behavioral responses in models of neuropathic pain (Lu et al., 2017). The gene discussed is KCNN4; the disease is Pain.