MALAT1 and nasopharyngeal carcinoma: MiR-483-5p directly decreases death-associated protein kinase 1 (DAPK1) protein expression, increases colony formation of NPC cells, reduces radiation-induced apoptosis and DNA damage, via activating the ERK signal pathway.137 On the contrary, miR-124 can inhibit the occurrence of NPC through the MALAT1/ERK/MAPK axis.138 In addition, Peng et al.139 found that the suppression of proliferation, invasion, and metastasis of NPC by miR-124 is achieved by regulation of Homo sapiens forkhead box Q1 (Foxq1).