ICAM1 and Sepsis: All sepsis cases showed a strong VLA-4 expression in intravascular, interstitial and intra-alveolar pulmonary leukocytes, unlike the non-sepsis control cases where immunoreactivity was weak in interstitial leukocytes and absent in intravascular and intra-alveolar leukocytes; ICAM-1 was strongly expressed in endothelial cells of pulmonary vessels, pulmonary macrophages, and lymphocytes in all sepsis but not in the non-sepsis control cases.