In accordance with our MS data, dysfunctions in glutamate recycling processes were implicated in AD and drastic declines in the excitatory amino acid transporter 1 (EAAT1) of astrocytes within human tissue and astrocytic cultures exposed to Aβ oligomers have been reported (Huang et al., 2018; Zoia et al., 2004). The gene discussed is SLC1A3; the disease is Alzheimer disease.