To test this hypothesis, weperformed single-round HIV infection using a VSV-G/HIV construct (which enterscells by endocytosis independently of CD4 and co-receptors [49]), cultured cells in the presence orabsence of T0070907, and analyzed by FACS the expression of BST-2 protein on thesurface of HIV-infected cells identified using HIV-Flow (Figure 7A), as previously reported [62]. The gene discussed is CD4; the disease is HIV infectious disease.