Concerning the potential function of KLK7 in ovarian cancer, Dong et al. [23] reported that KLK7 promotes multicellular aggregates (MCA) and α5/β1 integrin-dependent cell adhesion, which induce ovarian cancer cell invasion, metastasis, and paclitaxel resistance [46, 47], thereby increasing peritoneal dissemination and reinvasion. This evidence concerns the gene KLK7 and ovarian cancer.