In a model of experimental autoimmune myocarditis induced by immunizing BALB/c mice, a strain possessing a normal Pla2g2a gene, with the murine α-myosin heavy chain [30], PLA2R1 deficiency markedly increases sPLA2-IIA and -IB proteins (but not mRNAs) in the myocardium, probably as a result of their impaired clearance [35]. Here, PLA2G2A is linked to autoimmune myocarditis.