According to the report, SNHG17 is upregulated in CRC tissues and leads to enhancement of the CRC cell proliferation through knocking down P57, suggesting expression of SNORA71A is upregulated in CRC ([34]), whereas the recent study shows the transcription of SNORA71A in NSCLC tissues is independent from its host gene SNHG17 [29]. This evidence concerns the gene SNHG17 and colorectal carcinoma.