The analysis revealed that the levels of sICAM-1, IFN-γ, IL-1rα, IL-7, IL-17, CXCL1, M-CSF, CCL2, CCL5, CXCL12, TIMP-1 and TNF-α were increased to varying degrees upon CGS-17 and CXZ-15 infection under natural conditions (Fig. 2a–d). The gene discussed is CCL5; the disease is infection.