In allergic asthma, allergen induces interleukin‐4 (IL‐4) increased secretion by lung cells of the innate immune system, like innate lymphoid cells type 2, resulting in chronic differentiation of Th2 cells, eosinophil recruitment into the lung, induction of both memory B cells and long‐lived plasma cells producing high‐affinity IgE antibodies that bind to the high‐affinity receptor expressed on the surface of mast cells, basophils, and eosinophils.1 The gene discussed is IL4; the disease is allergic asthma.