Similar to other chemokines, CCL5 causes migration, invasion, and epithelial-to-mesenchymal transition (EMT) of cancer cells via its receptor [62,64,76,77], which is related to the activation of hedgehog [78], Wnt/β-catenin [70,79], and the Akt/PKB→nuclear factor κB (NF-κB) pathways [80,81,82]. This evidence concerns the gene CCL5 and cancer.