INS and metabolic syndrome: Nonetheless, we assume that GABAergic RIP-Cre25Mgn neurons in the ARC and/or DMH are key for anti-dyslipidemia actions of leptin in insulin-deficient mice, because (i) leptin-responsive GABAergic neurons are located only in the ARC, DMH, and LHA, and (ii) GABAergic RIP-Cre25Mgn neurons are anatomically limited to the ARC, DMH, and MTu; therefore, ARC and DHM are only overlapped regions that match to the anatomical and chemical-classification profiling from previous studies.