Pathway analyses of these alterations revealed activation of peroxisome proliferator-activated receptor alpha (PPAR-alpha)/ Retinoid X receptor alpha (RXR-alpha) as well as cyclin-dependent kinase 5 (CDK5) signaling, but inhibition of metabolic pathways, namely Oxidative Phosphorylation, in OCCC vs HGSOC patient tumor tissues (Supplementary Data 5). The gene discussed is CDK5; the disease is neoplasm.