In breast cancer, apoptosis following paclitaxel treatments is boosted by miR-7-5p, miR-542-3p, miR-621, and miR-5195-3p via directly modulating BCL-2, survivin, F-box protein 11 (FBXO11), and eukaryotic translation initiation factor 4A2 (EIF4A2), respectively [307,321,325]. Here, BCL2 is linked to breast carcinoma.