In glioma, miR-92a has been identified to accelerate proliferation, cell survival, and metastasis through regulating BCL2L11, cadherin 1 (CDH1, also known as E-cadherin) and the Akt/mechanistic target of rapamycin kinase (mTOR) signaling [118,119,120]. Here, AKT1 is linked to central nervous system cancer.