The results showed that overexpressing STK3 significantly affected multiple inflammatory responses, including I-kappaB kinase/NF-kappa B signaling, I-kappa B kinase/NF-kappa B signaling complex, CXCR chemokine receptor binding, and NF-kappa B signaling pathway, indicating a regulatory role of STK3 in ovarian cancer immune microenvironment (Figure 4(a)). This evidence concerns the gene STK3 and ovarian carcinoma.