TTP (i.e., GA-VMTD; AA-VMTD) utilizes activated “aberrant” ULVWF path due to ADAMTS13 deficiency and produces disseminated microthrombosis (i.e., DIT), and APL with consumption coagulopathy (true DIC) utilizes “aberrant” TF path due to abnormally expressed TF from leukemic promyelocytes and produces disseminated fibrin clot disease. Here, TF is linked to acute promyelocytic leukemia.