NFKB1 and asthma: Galangin attenuated in vitro and in vivo airway remodeling by inhibiting TGF-β1-mediated ROS generation and MAPK/Akt phosphorylation in asthma; also, galangin improved ovalbumin (OVA)-induced airway inflammation by inhibiting NF-κB pathway, which reduced eosinophils, neutrophils, and lymphocytes infiltration and goblet cell hyperplasia.