(b) Wu et al. deciphered the MALAT1/miR-608/HOXC4 axis in UM, showing that MALAT1 is capable of enhancing HOXC4 expression through the molecular interaction and inhibition of miR-608, ultimately promoting in vitro UM cell proliferation, invasion, and migration and in vivo tumor growth [104] (Figure 2). The gene discussed is MALAT1; the disease is neoplasm.