MMP3, also known as stromelysin-1, was dramatically increased in the blood circulation following GC treatment, as previously shown by us [17] and others in both DMD patients [24] and inflammatory bowel disease patients [25] and anti-neutrophil cytoplasmic antibody-associated vasculitis, and juvenile dermatomyositis [25]. The gene discussed is MMP3; the disease is Duchenne muscular dystrophy.