SELP and myocardial infarction: Nonselective inhibition of inflammation, such as corticosteroids and nonsteroidal anti-inflammatory drugs 8, selective targeting of pro-inflammatory signals, such as CD11/CD18 integrin inhibition 34, and P-selectin inhibition 35, after MI might have adverse effects on scar formation and myocardial remodeling, thereby raising the risk of cardiac rupture.