Etomoxir, an FAO inhibitor, blocked IL-4-induced M2 macrophage polarization [189], and EI-05, activator of the intracellular lipid chaperone E-FABP (epithelial fatty acid binding protein), led to the increase in IFN-β production by macrophages and improved antitumoral responses in murine models of breast cancer [190]. Here, IL4 is linked to breast carcinoma.