We next established an in vivo model to investigate if CCL3/CCL4-secretion influences endogenous leukocyte recruitment to tumours engrafted in mice (Figure 4—figure supplement 4), and showed that CCL3/CCL4-secreting tumours consistently recruit more endogenous NK cells than contralateral control tumours (Figure 4—figure supplement 4B). The gene discussed is CCL4; the disease is neoplasm.