Furthermore, IFNγ, produced by activated CTLs and abundant in inflamed tumours, can induce CXCL9 and CXCL10 secretion in various intermediary cells, including tumour-infiltrating myeloid cells (Dobrzanski et al., 2001; Gordon-Alonso et al., 2017; Hickman et al., 2015), which may enhance the role of CXCR3 in homing. This evidence concerns the gene CXCR3 and neoplasm.