Recent findings also support the notion that about 15%-20% of peripartum heart failure patients have cardiomyopathy-induced mutations, i.e., in genes such as titin, beta-myosin, myosin-binding protein C, lamin A/C, or sodium voltage-gated channel alpha subunit 5 (SCN5A). The gene discussed is SCN5A; the disease is cardiomyopathy.