NFKB1 and colitis: GA could inhibit inflammation in dextran sulfate sodium (DSS)-induced colitis in mice through the suppression of p65-NF-κB and IL-6/p-STAT3Y705 activation (22), and suppress lipopolysaccharide (LPS)-induced inflammation in transgenic mice by regulating immune system processes and downregulating the NF-κB pathway (97).