IL6 and Feingold syndrome: The levels of proinflammatory cytokines, such as interleukin-6 (IL-6) and tumor necrosis factor-α (TNF-α), and anti-inflammatory cytokines, such as interleukin-4 (IL-4), in the peripheral blood of children with FS also exhibited significant changes, suggesting that these inflammatory molecules may play an important role in the pathogenesis of FS (8–10).