UBE2I and acute myocardial infarction: Our data from Ubc9 overexpression and Ubc9 silencing tests indicate that endogenous Ubc9 is beneficial to ischemic cardiomyocytes, and when exogenous Ubc9 is applied, the apoptosis of the hypoxic cardiomyocytes is significantly reversed, which is critical at the beginning of the repair process after acute myocardial infarction.