Besides, a decreased expression of miR-29a has also been observed in other inflammatory conditions, such as in fibroblasts in systemic sclerosis [20], in lung and plasma in chronic obstructive pulmonary disease [21], in renal tissues in IgA nephropathy [22], in peripheral blood T cells in Hashimoto’s thyroiditis patients [23], and upon bacterial infection in IFN-γ producing cells [24]. This evidence concerns the gene IFNG and chronic obstructive pulmonary disease.