FOXM1 and glioblastoma: Increased ALKBH5 indicates poor prognosis of patients with glioblastoma while ALKBH5 depletion interferes with self‐renewal ability and inhibits the proliferation and tumorigenesis of GSCs by sustaining FOXM1 expression via ALKBH5‐mediated m6A demethylation of FOXM1 nascent transcripts.72