IL12A and infection: Among the few specific responses, we observed higher levels of IL-12A and IL-12B gene expression and IL-12-dependent regulation upon infection with C. glabrata and C. parapsilosis, in agreement with previous data showing a lack of or even active repression of IL-12 production by C. albicans (51, 52) and a higher level of IL-12 release during whole-blood infection with C. glabrata than with C. albicans (53).