IL4 and infection: Considering that TCRγδ+ cells also promote epithelial repair [38], it can be postulated that in our model, the role of these cells would be to act as a source of cytokines at the beginning of the infection (coincident with the increase in IL-4 levels, Fig. 8) and promoting, along with M2 macrophages, the repair of the T. spiralis-damaged epithelium.