However, earlier evidence [25] only revealed that inhibited miR-92a could promote the WNT1-inducible signaling pathway protein 1 (WISP1) in lung fibroblasts, a fibrogenic medium, but they did not further analyze fibroblast changes, so miR-92a may inhibit the development of pulmonary fibrosis. Here, CCN4 is linked to pulmonary fibrosis.