CXCL2 (MIP‐2) and CXCL1 (KC), which are responsible for the infiltration of neutrophils and monocytes, were suppressed at the beginning of infection (6 h), whereas inhibition of CCL2 (MCP‐1), TNF‐α, IL‐6, IL‐1β and IL‐17A appeared after 24 h. The gene discussed is IL17A; the disease is infection.