C. asiatica and its triterpenoids affect neurological diseases possibly through the mitogen-activated protein kinase (MAPK) signaling pathway, phosphotidylinositol 3 kinase/protein kinase B/mammalian target of rapamycin (PI3K/Akt/mTOR)signaling pathway, and nuclear factor kappa-light-chain-enhancer of activated B cells (NF-kB) signaling pathway (Table 2). This evidence concerns the gene AKT1 and nervous system disorder.