NLRC5 acts as an inhibitor of inflammation in ovarian endometriosis by inducing autophagy: Over-expression of NLRC5 could promote the expressions of LC3, Beclin1, and autophagosomes formation in EESCs, thereby inhibiting IL-6 and TNF-α expressions in ovarian endometriosis, suggesting that promoting NLRC5 and autophagy may be novel therapeutic methods in ovarian endometriosis (Figure 7). This evidence concerns the gene IL6 and ovarian endometriosis.