CSNK2B and glioblastoma: Further, this study also showed that depletion of CSNK2β modulated molecular markers associated with apoptosis and autophagy, which is consistent with earlier reports of involvement of CSNK2 in suppressing apoptosis, as overexpression of CSNK2 in prostate cancer cells prior to treatment with etoposide rescued against cell death (Guo et al., 2001[18]) and downregulation of CSNK2 (α subunit) leads to autophagic cell death in glioblastoma cell lines (Olsen et al., 2012[38]).