BNIP3 and myocardial infarction: Furthermore, in vitro studies demonstrated a cardioprotective role of miRNA-210 in H2O2-induced cardiomyocyte apoptosis by regulating the pro-apoptotic Bcl-2 adenovirus E1B 19kDa-interacting protein 3 (BNIP3) [69], as well as it being implicated in the mechanisms of hypoxic-mesenchymal-stem-cell-derived exosomes for cardiac repair after myocardial infarction [70].