Therefore, it would be tempting to speculate that a more specific separation of EVs using CD44 as a target antigen, and thereby enriching the population for glioblastoma EVs, would lead to a correlation of high miR-328 levels with a lower prognosis, as was the case in our study and as reported by Delic et al. [44]. The gene discussed is CD44; the disease is glioblastoma.