Of note: the study in which the genome-wide significant association with AD of the variant in/near KANSL1 was originally identified, reported a larger effect-size compared to the effect-size used in our manuscript (β = 0.31 and β = 0.07, respectively), possibly because the original analysis was stratified by APOE. We cannot exclude that we underestimated the contribution of KANSL1 in the analyses. The gene discussed is APOE; the disease is Alzheimer disease.