These findings support and extend our previous observations that neither the selective depletion of HSPA2 nor double knockdown of HSPA2 and HSPA1A/B expression decreased the proliferation or viability of NSCLC cells (previously demonstrated in NCI-H1299 and NCI-H23 cell lines) [18]. The gene discussed is HSPA1A; the disease is non-small cell lung carcinoma.