IL17A and Peritoneal Fibrosis: An early study showed that, in a murine model of chronic exposure to conventional PDF leading to peritoneal fibrosis [111], the immune response was activated; was characterized by peritoneal infiltration of Th17 and γδ T cells but no Th1 and Th2 cells; increased peritoneal expression of Th17-related cytokines, such as IL-17A and IL-6; and activated Th17-related transcription factors, including RORγt and STAT3 [35].