The bioinformatics findings of this study suggested that the specific inactivation of signaling pathways of inflammatory stress and targeted modulation of intrapulmonary EGFR, CASP3, STAT3, ESR1, FPR2, F2, BCL2L1, BDKRB2, MPO, and ACE expressions, maybe potential pathways of TJQW action against COVID-19. Here, BCL2L1 is linked to COVID-19.