Data from an earlier study on lung adenocarcinoma [12] showed that miR-98 expression negatively correlated with LINC00665 expression and that the latter acted as a ceRNA to competitively bind miR-98, thereby activating the AKR1B10–ERK-signaling pathway to facilitate cell proliferation and metastasis. This evidence concerns the gene AKR1B10 and lung adenocarcinoma.